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INHIBITION OF APOPTOSIS BY bcr-abl FUSION GENE IN K562 CELLS

   
@article{CJCR1661,
	author = {Chun-hong Wang and Bing-zhong Sun and Yue-chuan Yuan},
	title = {INHIBITION OF APOPTOSIS BY bcr-abl FUSION GENE IN K562 CELLS},
	journal = {Chinese Journal of Cancer Research},
	volume = {11},
	number = {3},
	year = {2013},
	keywords = {},
	abstract = {Objective: To investigate the effect of bcr-abl fusion gene on CML cell apoptosis. 
Methods: Apoptosis of exvivo cultured K562 cells were observed after exposure to synthetic 18 mer antisense oligodeoxynucleotide complementary to the bcr-abl junction (b3a2). 
Results: Apoptosis of K562 cells was significantly increased associated with inhibition of bcr-abl expression. 
Conclusion: bcr-abl fusion gene formation due to chromosome translocation may be the major mechanism of CML via inhibition of apoptosis.},
	issn = {1993-0631},	url = {https://cjcr.amegroups.org/article/view/1661}
}